DANIEL FUENTES MOREIRA
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- Memory in children with temporal lobe epilepsy is at least partially explained by executive dysfunction(2012) RZEZAK, Patricia; GUIMARAES, Catarina A.; FUENTES, Daniel; GUERREIRO, Marilisa M.; VALENTE, Kette D.An association between memory and executive dysfunction (ED) has been demonstrated in patients with mixed neurological disorders. We aimed to investigate the impact of ED in memory tasks of children with temporal lobe epilepsy (TLE). We evaluated 36 children with TLE and 28 controls with tests for memory, learning, attention, mental flexibility, and mental tracking. Data analysis was composed of comparison between patients and controls in memory and executive function; correlation between memory and executive function tests; and comparison between patients with mild and severe ED in memory tests. Children with TLE had worse performance in focused attention, immediate and delayed recall, phonological memory, mental tracking, planning, and abstraction. Planning, abstraction, and mental tracking were correlated with visual and verbal memory. Children with severe ED had worse performance in verbal and visual memory and learning tests. This study showed that ED was related to memory performance in children with TLE.
conferenceObject Correlation between novelty-seeking and harm-avoidance with depression and anxiety in Parkinson's disease(2012) FONOFF, F. M. C.; FONOFF, E. T.; QUARANTA, T.; BARBOSA, E. R.; TEIXEIRA, M. J.; FUENTES, D.Background: Lower novelty-seeking (NS) and higher harm-avoidance (HA) are characteristics often found in depressive patients. Depression is a comorbidity often found in PD patients. However, recent studies debate whether depressive-like personality traits in PD patients result from dopaminergic degeneration or are related to depression symptoms present in PD. Methods: 61 advanced PD patients and 42 healthy volunteers (HV) were submitted to personality assessment through Temperament and Character Inventory by Cloninger and to depressive and anxiety symptoms assessment through Hamilton Scales for Anxiety (Ham-A) and Depression (Ham-D). Results: PD group showed NS1 significantly lower 4,96 (SD: 2,05) and higher HA4 4,96 (SD: 1,72) than HV. However, PD group and HV did not differ significantly in Anxiety and Depression scores. Conclusions: The present data suggest that depression is not responsible for depressive-like features in PD. Following this reasoning, we can state that these personality traits can be understood independently and, therefore, as part of the motor treatment of the PD patients, a clinical follow-up of the personality traits is necessary. Further studies should investigate those personality traits in PD patients after DBS.