GABRIELA NATANIA SALES REBELO

(Fonte: Lattes)
Índice h a partir de 2011
2
Lattes
ORCID
Projetos de Pesquisa
Unidades Organizacionais
Instituto Central, Hospital das Clínicas, Faculdade de Medicina
LIM/45 - Laboratório de Fisiopatologia Neurocirúrgica, Hospital das Clínicas, Faculdade de Medicina

Filtros

Limpar filtros

Configurações

Resultados de Busca

Agora exibindo 1 - 2 de 2
  • article 10 Citação(ões) na Scopus
    Syntopy of vagus nerve in the carotid sheath: A dissectional study of 50 cadavers
    (2019) HOJAIJ, Flavio; REBELO, Gabriela; AKAMATSU, Flavia; ANDRADE, Mauro; CAMARGO, Cristina; CERNEA, Claudio; JACOMO, Alfredo
    Background Vagus nerve anatomical position inside the carotid sheath is not clear in the literature. Nevertheless, monitoring laryngeal nerves during thyroid surgeries may damage big vessels in the carotid sheath (jugular vein; carotid artery). This gap led to an unprecedent cross sectional study of vagus syntopy using the carotid artery as anatomical mark. Methods Fifty cadavers less than 24 hours postmortem were studied. The vagus nerve was spotted, reproducing the patterns performed in thyroidectomies. Results On the right side, vagus nerve was posterior to the common carotid artery in 64% of the cases. On the left side, it was anterior, in 68% of the dissections. Comparing both sides, there was no symmetry in this syntopy. No influence of ethnic or anthropometric characteristics was observed. Conclusion The vagus nerve is more frequently posterior to the common carotid artery on the right side and, anterior, on the left side. Level of Evidence 4
  • article 31 Citação(ões) na Scopus
    Genetic analysis of patients with familial and sporadic amyotrophic lateral sclerosis in a Brazilian Research Center
    (2017) CHADI, Gerson; MAXIMINO, Jessica Ruivo; JORGE, Frederico Mennucci De Haidar; BORBA, Fabricio Castro De; GILIO, Joyce Meire; CALLEGARO, Dagoberto; LOPES, Camila Galvao; SANTOS, Samantha Nakamura Dos; REBELO, Gabriela Natania Sales
    Objective: To investigate gene mutations in familial form (FALS) and sporadic form (SALS) of amyotrophic lateral sclerosis (ALS) in a highly miscegenated population. Methods: Frequencies of mutations in the C9orfF72, TARDBP, SOD1, FUS and VAPB genes were investigated in a cohort of FALS (n=39) and SALS (n=189) subjects from the Research Centre of the University of SAo Paulo School of Medicine. All patients were subjected to C9orf72 and TARDBP analyses. SOD1, FUS and VAPB were also evaluated in FALS subjects. Results: Mutations were identified in FALS (61.3%) and SALS (5.3%) patients. Mutations in C9orf72 (12.8%,>45 GGGGCC hexanucleotide repeats), VAPB (43.6%, P56S) and SOD1 (7.7%, L145S) were identified in FALS subjects. Pathogenic C9orf72 expansions (2.64%) were identified in some SALS patients. Similar changes of TARDBP were found in SALS (2.64%) but not in FALS subjects. No FUS mutations were seen in any FALS subjects. Conclusions: TARDBP and C9orf72 mutations in this cohort were similar to those found in other centres worldwide. VAPB mutation (P56S) was highly prevalent in Brazilian FALS patients.