Livros e Capítulos de Livros - LIM/53
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A coleção de Livros e Capítulos de Livros reúne capítulos e resumos de obras produzidas por autores do sistema FMUSP-HC que inclui a Faculdade de Medicina da Universidade de São Paulo (FMUSP), o Hospital das Clínicas da FMUSP e demais institutos associados.
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bookPart Diagnóstico laboratorial e radiológico(2021) GIANNINI, Maria José Soares Mendes; NEGRO, Gilda Maria Barbaro Del; GONçALVES, Elmar Gonzaga- 3.41 - Skin Barrier, Microbiome and Psoriasis(2022) HIRAYAMA, A. L. S. da; FONSECA, D. C.; ROMITI, R.Psoriasis is an inflammatory skin disorder of multifactorial etiology related to dysregulation of innate and adaptive immune responses, genetic inheritance and environmental factors. Skin and gut share features and functions. Each one has a singular ecosystem that interacts with epithelial and immune responses. The association of psoriasis with inflammatory bowel disease is well-documented, as well as immune and correlated inflammatory pathways. Recent studies have shown differences in the microbiome of psoriasis suggesting that the influence of gut and skin microbiome can possibly be related with the pathophysiology, disease's course and even its prognosis and treatment response. © 2022 Elsevier Inc. All rights reserved.
- Dermatological Manifestations(2023) CURY-MARTINS, J.; SANCHES, J. A.Cutaneous involvement by amyloidosis is characterized by extracellular deposition of amyloid protein within the skin and appendages and can occur as a skin-limited disorder (localized cutaneous amyloidosis—LCA) or as a manifestation of systemic amyloidosis (SA). LCA can be divided into primary LCA—macular, lichen and nodular amyloidosis; or secondary LCA. The main subtypes of SA with cutaneous involvement include immunoglobulin light chain (AL) amyloidosis, secondary (AA) amyloidosis, dialysis-related amyloidosis, and heredofamilial amyloidosis. Skin involvement is present, for example, in approximately 40% of patients with AL amyloidosis, and might provide an early clue to the existence of an underlying plasma-cell dyscrasia. © The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerland AG 2023.
bookPart Doenças da unha(2023) CESAR, Denise Vieira Galvão; CESAR, Wagner G. Galvão; CHIACCHIO, Nilton Gioia DibookPart Apresentação da 4ª edição(2015) FESTA NETO, Cyro; CUCé, Luiz Carlos; REIS, Vitor Manoel Silva dosbookPart Apresentação(2019) FESTA NETO, Cyro; CUCé, Luiz Carlos; REIS, Vitor Manoel Silva dos- Paracoccidioidomycosis: An endemic mycosis in the Americas(2015) TABORDA, C. P.; J, M. E. Uran; TRAVASSOS, L. R.Paracoccidioidomycosis (PCM), is a systemic mycosis with clinical manifestations of a granulomatous disease, caused by thermally dimorphic Paracoccidioides spp. Adolpho Lutz first described it in 1908, while examining oral lesions in two patients. The first patient, a Spanish man, 40 years old, reported oral lesions for six months and the second patient, a 30 year old man, reported a four-year history of multiple oral lesions. Five months later, Lutz reported that the patient returned quite emaciated, with hoarseness and diarrhea (Reviewed by Marques 2008). Primarily named as “pseudococcidial hyphoblastomycosis” (pseudococcidial to differentiate from coccidioidomycosis and hifoblastomycosis), the mycosis was then called South American Blastomycosis or Lutz and Splendore-Almeida disease (Lacaz et al. 1991). The actual term Paracoccidioidomycosis was established in 1971 in Medellín, Colombia, during a meeting of the American Continent mycologists and has been widely accepted since then (Lacaz 1982). © 2016 by Taylor & Francis Group, LLC.
- Paracoccidioidomycosis: Advance towards a molecular vaccine(2013) TRAVASSOS, L. R.; RITTNER, G. M. G.; TABORDA, C. P.The P10 peptide is a conserved sequence in the gp43 diagnostic antigen of most isolates of Paracoccidioides brasiliensis. P10 is promiscuously presented by three different MHC class II molecules from mouse haplotypes, and also by 90 % of Caucasian HLA-DR antigens. It elicits an IFN-γ-dependent protective anti-fungal response and is a candidate for a peptide vaccine. Immunization with P10 of mice intratracheally challenged with virulent yeasts rendered significant protection measured by colony-forming units in the lungs. A predominant Th-1 immune response was obtained. Different adjuvants enhanced the activity of the peptide and P10-primed dendritic cells were highly effective in clearing fungal infection. Gene therapy with plasmids expressing P10 and IL-12 elicited a protective Th-1 immune response without causing hyperinflammation or fibrosis. In a long-term experiment, vaccination with pP10 and pIL-12 virtually eliminated all fungal elements and preserved a normal lung parenchyma. © 2013 Springer-Verlag Wien. All rights reserved.
bookPart Dermatite da área das fraldas(2022) FERNANDES, Juliana Dumêt; OLIVEIRA, Zilda Najjar Prado debookPart Dermatoses do recém-nascido(2022) BESSA, Vanessa Rolim; SAMORANO, Luciana de Paula; OLIVEIRA, Zilda Najjar Prado debookPart Afecções fisiológicas da pele do recém-nascido(2022) BESSA, Vanessa Rolim; SAMORANO, Luciana de Paula; OLIVEIRA, Zilda Najjar Prado debookPart Infestações e infecções de pele(2022) ROTTER, Anita; OLIVEIRA, Zilda Najjar Prado debookPart Fotoproteção(2022) MIOTTO, Isadora Zago; SAMORANO, Luciana de Paula; OLIVEIRA, Zilda Najjar Prado debookPart Principais dermatoses na infância(2022) SAMORANO, Luciana de Paula; OLIVEIRA, Zilda Najjar Prado debookPart Dermatoses papulosas e pruriginosas(2022) MARUTA, Celina Wakisaka; ORFALI, Raquel LeãobookPart Infecções sexualmente transmissíveis: sífilis, HPV, HIV e oftalmite gonocócica(2022) MIOTTO, Isadora Zago; OLIVEIRA, Walmar Roncalli Pereira de; FESTA NETO, CyrobookPart Nevos melanocíticos(2022) MIYASHIRO, Denis Ricardo; SANCHES JR., José AntoniobookPart Cabelos(2019) VALENTE, Neusa Yuriko SakaibookPart Dermatoses paraneoplásicas(2019) BERG, Roberta Vasconcelos; MIYASHIRO, DenisbookPart Pápula(2019) MARUTA, Celina Wakisaka