Livros e Capítulos de Livros - FM/MCM

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A coleção de Livros e Capítulos de Livros reúne capítulos e resumos de obras produzidas por autores do complexo Hospital das Clínicas - Faculdade de Medicina da Universidade de São Paulo.

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  • bookPart
    Tele-educação física pediátrica
    (2023) ASTLEY, Camilla; SILVA, Clovis Artur Almeida da; GUALANO, Bruno
  • bookPart
    Síndrome cardiorrenal
    (2023) ASSIS, Luiza Liza de; JORGE, Letícia Barbosa; SOFFIATTI, Carla David; YU, Luis
  • bookPart
    Choque séptico
    (2023) JúNIOR, Armindo Jreige; SILVA, Mariana Furtado; HAJJAR, Ludhmila Abrahão
  • bookPart
    Emergências cardiovasculares no paciente com câncer
    (2023) ANDRADE, Fernanda Thereza de Almeida; BITTAR, Cristina Salvadori; SHINZATO, Mariane Higa; NAKADA, Letícia Naomi; BARROS, Cecília Chie Sakaguchi; HAJJAR, Ludhmila Abrahão
  • bookPart
    Hipertensão pulmonar
    (2023) PARENTE, Yuri de Deus Mont´Alverne; JúNIOR, José Leonidas Alves; HAJJAR, Ludhmila Abrahão
  • bookPart
    Fluxo de atendimento do cardiopata na emergência
    (2023) RIBEIRO, Lucas Trindade Cantú; MELO, Rafael Yuji; PINESI, Henrique Trombini; SCALABRINI NETO, Augusto
  • bookPart
    Apresentação
    (2023) HAJJAR, Ludhmila Abrahão
  • bookPart
    Choque cardiogênico
    (2023) KAWAHARA, Lucas Tokio; BARROS, Cecília Chie Sakaguchi; NAKADA, Letícia Naomi; HAJJAR, Ludhmila Abrahão
  • bookPart
    Dor em reumatologia
    (2022) FULLER, Ricardo; SHINJO, Samuel Katsuyuki; KOBAYASHI, Carolina Besser Cozac
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  • bookPart
    Aids: manifestações hematológicas e oncológicas relacionadas
    (2021) VELLOSO, Elvira Deolinda Rodrigues Pereira; PRACCHIA, Luis Fernando; SILVA JUNIOR, Wellington Fernandes da
  • bookPart
    Sangramento, anemia e transfusão
    (2023) BARROS, Cecília Chie Sakaguchi; KAWAHARA, Lucas Tokio; VEIGA, Alicia Dudy Muller; HAJJAR, Ludhmila Abrahão
  • bookPart
    Curso experimental de medicina da USP, agressões e legado
    (2023) MACHADO, Marcello Marcondes
  • bookPart
    Monitorização hemodinâmica
    (2023) NAKADA, Letícia Naomi; QUINTãO, Vinícius Caldeira; CARMONA, Maria José Carvalho; HAJJAR, Ludhmila Abrahão
  • bookPart 0 Citação(ões) na Scopus
    46,XY differences of sex development (DSD) due to 17β-hydroxysteroid dehydrogenase type 3 deficiency
    (2023) GOMES, N. L.; COSTA, E. M. F.; INACIO, M.; MARTIN, R. M.; NISHI, M. Y.; CARVALHO, F. M.; SIRCILLI, M. H. P.; TIBOR, F. D.; DOMENICE, S.; MENDONCA, B. B.
    In this chapter, we revise the epidemiological, clinical, hormonal, genetical findings and also the long-term outcomes of 46,XY individuals with 17β-HSD3 deficiency based on the review of previously reported cases and also our own cases. © 2023 Elsevier Inc. All rights reserved.
  • bookPart 1 Citação(ões) na Scopus
    Urological and Kidney Involvements in Amyloidosis
    (2023) FEITOSA, V. A.; ONUCHIC, L. F.; NORONHA, I. de Lourdes
    Amyloidosis is a group of disorders in which soluble proteins aggregate and deposit extracellularly in tissues as insoluble fibrils, causing organ dysfunction. Clinical management depends on the subtype of deposited protein and the affected organs. Systemic amyloidosis may stem from anomalous proteins such as immunoglobulin light chains and serum amyloid proteins in chronic inflammatory settings or derive from hereditary disorders. The kidney is the organ most commonly involved in systemic amyloidosis. Renal amyloidosis is characterized by acellular pathologic Congo red-positive deposition of amyloid fibrils in glomeruli, vessels, and/or interstitium. This disease manifests with heavy proteinuria, nephrotic syndrome, and/or progression to end-stage kidney failure. In addition to renal involvement, familial amyloidotic polyneuropathy often leads to urological dysfunction, and primary localized amyloidosis has been reported in the genitourinary tract, including limited deposition in the renal pelvis, ureters, or bladder. © The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerland AG 2023.
  • bookPart 0 Citação(ões) na Scopus
    Idiopathic Short Stature: Diagnostic and Therapeutic Approach
    (2022) CANTON, A. P. M.; JORGE, A. A. L.
    Our clinical case for discussion is about a short statured boy who was 14.6 years old at his first evaluation. He was born after a 39-week gestation as the third child of a non-consanguineous marriage. His birth weight was 3.250 g (-0.3 SDS), and his birth length was not available. His neuropsychomotor development was normal, his school performance was good, and there were no remarkable findings in his medical history. His father’s height was 174 cm (-0.1 SDS), and his mother’s height was 154 cm (-1.3 SDS), resulting in a target height of 170.6 cm (-0.6 SDS). His father and mother apparently had normal pubertal development, and his mother’s age of menarche was 13 years old. His older brother’s height was not available, but he had a previous history of pubertal spurt after 16 years of age. Likewise, his older sister’s height was also not available, but she had a previous history of menarche at 14. © Springer Nature Switzerland AG 2022.
  • bookPart 0 Citação(ões) na Scopus
    DO YOU HAVE TO LIE TO INDUCE PLACEBO EFFECTS?: The use of open label placebos in sport and exercise
    (2023) SAUNDERS, B.; MARTICORENA, F. M.; GUALANO, B.
    The use of deceptive placebo in practice is inherently fraught with ethical constraints and trust issues, meaning that alternative methods must be employed in practice. This has led to the development of an alternative approach through which placebo effects might be generated, namely open-label placebo (also called “honest placebo”). This method consists of openly informing the individual that they are receiving an inert intervention (i.e., a placebo). While there is a growing body of evidence demonstrating the efficacy of this method in clinical practice, data in sport and exercise remain scarce. In this chapter, we discuss the use of open-label placebo as a potential ergogenic intervention in the context of exercise and sports, we propose theoretical mechanisms through which they work, and we place an emphasis on practical applications and research gaps in this emerging area of research. © 2024 selection and editorial matter, Philip Hurst and Chris Beedie; individual chapters, the contributors.
  • bookPart 0 Citação(ões) na Scopus
    Rheumatic fever: From pathogenesis to vaccine perspectives
    (2023) GUILHERME, L.; BRANCO, C. E.; BARROS, S. F. De; KALIL, J.
    Rheumatic fever (RF) is considered a model of autoimmune disease due to untreated throat infection by S. pyogenes that affects children and teenagers. The autoimmune process is believed to be the basis of all of the clinical manifestations; for instance, arthritis by immune complex deposition, chorea by antibody binding to neuronal cells, skin and subcutaneous manifestations that are mediated by a delayed hypersensitivity reaction, and carditis that is caused by cross-reactive antibodies and T cells. This chapter presents an overview of the mechanisms leading to the tissue lesions, treatment, and future possibilities of a vaccine against S. pyogenes. © 2023 Elsevier Inc. All rights reserved.