CAIO REBOUCAS FONSECA CAFEZEIRO

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  • conferenceObject
    Myocardial Stiffness evaluation by shear wave elastography in transthyretin amyloidosis with and without cardiac involvement
    (2023) ALENCAR NETO, A. C. Comte de; CAFEZEIRO, C. R. F. C. Caio Reboucas Fonseca; BUENO, B. V. K. B. Bruno Vaz Kerges; RISSATO, J. H. R. Joao Henrique; HOTTA, V. T. H. Viviane Tiemi; ALENCAR FILHO, A. C. A. F. Aristoteles Comte De; PEREIRA, F. L. P. Fernando Linhares; COUCEIRO, K. C. Katia; ROCHITTE, C. E. R. Carlos Eduardo; BARBOSA, J. M. B. B. Joao Marcos Bemfica; SOARES JUNIOR, J. S. J. Jose; RAMIRES, F. J. A. R. Felix Jose Alvarez; KALIL FILHO, R. K. F. Roberto; MATHIAS JUNIOR, W. M. J. Wilson; FERNANDES, F. F. Fabio
  • conferenceObject
    Coronary flow reserve by PET 13N-ammonia in patients with hereditary transthyretin amyloidosis with and without cardiac involvement
    (2022) ALENCAR NETO, A. C.; CAFEZEIRO, C. R. F.; BUENO, B. V. K.; SOUZA, F. Ribeiro De; RISSATO, J. H. S. Henrique; BORGES, T. Souza; CARVALHAL, S. Freitas; LIMA, M. Santos; BUCHPIGUEL, C. Alberto; CHALELA, W. Azem; RAMIRES, F. J. Alvarez; SZOR, R. Shcolnik; KALIL FILHO, R.; ROCHITTE, C. E.; FERNANDES, F.
  • conferenceObject
    Aortic and Renal Artery Thrombosis as the First Clinical Manifestation of COVID-19 in a Heart Transplant Recipient
    (2021) BELFORT, D. S.; CAFEZEIRO, C. R.; FURLAN, D. A.; LIRA, M. S.; DANTAS, R. C.; ARAGAO, C. A.; CAMPOS, I. W.; AVILA, M. S.; MANGINI, S.; SEGURO, L. B.; MARCONDES-BRAGA, F. G.; BACAL, F.
  • bookPart
    Infecções oportunistas em pacientes transplantados
    (2022) CAFEZEIRO, Caio; QUINTANILHA, Nádia Romanelli
  • conferenceObject
    Non-invasive assessment of myocardial stiffness by the two-dimensional shear wave elastography ultrasound technique in patients with amyloidosis and Fabry disease
    (2023) CAFEZEIRO, C.; NETO, A. C. Alencar; BUENO, B. V. K.; RISSATO, J. H.; PEREIRA, N. M.; PEREIRA, F. L.; RAMIRES, F. J. A.; MATHIAS JR., W.; ROCHITTE, C. E.; HOTTA, V. T.; DABARIAN, A. L.; FERNANDES, F.
  • conferenceObject
    Routine screening practices for cardiac involvement in patients with sarcoidosis
    (2019) MOURA, D. M. M.; ARIMURA, F. E. A.; KAIRALLA, R. A. K.; ROCHITTE, C. E. R.; DABARIAN, A. D.; FEITOSA, M. F.; CAFEZEIRO, C. R. F. C.; SOARES, J. S.; RAMIRES, F. J. A. R.; HOTTA, V. T. H.; MADY, C. M.; FERNANDES, F. F.
  • article 1 Citação(ões) na Scopus
    Epipericardial fat necrosis as cause of chest pain in patient after heart transplantation
    (2020) CAFEZEIRO, Caio Reboucas Fonseca; LOPES, Mariana Pezzute; SILVA, Caio Tavares; AVILA, Monica Samuel; SEGURO, Luis Fernando B. C.; MANGINI, Sandrigo; CAMPOS, Iascara Wozniak; GAIOTTO, Fabio A.; MARCONDES-BRAGA, Fabiana G.; BACAL, Fernando
    Epipericardial fat necrosis is an uncommon clinical condition of unknown etiology. It typically presents as acute pleuritic chest pain and should be differentiated from acute pulmonary embolism and acute coronary syndrome. This condition is diagnosed by characteristic chest computed tomography findings of an ovoid mediastinal fatty lesion with intrinsic and surrounding soft-tissue stranding. Treatment of epipericardial fat necrosis includes the administration of anti-inflammatory agents, and symptoms usually resolve within a few days after treatment initiation. This disease entity has rarely been reported since it was first described in 1957. Most current knowledge of epipericardial fat necrosis is based on case reports that describe this condition in previously healthy individuals. We present the case of a 39-year-old woman with a history of heart transplant, who presented with chest pain secondary to epipericardial fat necrosis. Serial computed tomography revealed lesion resolution after appropriate treatment.
  • bookPart 0 Citação(ões) na Scopus
    Cardiologic Manifestation in Amyloidosis
    (2023) ALENCAR NETO, A. C. de; CAFEZEIRO, C. R. F.; BUENO, B. V. K.; SINGULANE, C. de Carvalho; FERNANDES, F.
    ATTR and AL amyloidosis are responsible for approximately 95% of all cases of CA. Cardiac amyloidosis (CA) is a relatively common cause of heart failure with preserved ejection fraction (HFpEF). It is important to know that extracardiac changes may precede cardiac involvement by a few years; thus, it is essential to recognize these findings as part of the clinical feature in a patient with amyloidosis. CA results from the progressive deposition of amyloid fibrils in the extracellular space. The involvement of amyloidosis includes mechanical and electrical manifestations. The most frequent CA clinical presentation is HFpEF with a restrictive pattern leading to diastolic dysfunction, followed by systolic dysfunction. Conduction disorders and arrhythmias are commonly associated. © The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerland AG 2023.
  • article 2 Citação(ões) na Scopus
    Non-invasive diagnosis of transthyretin cardiac amyloidosis utilizing typical late gadolinium enhancement pattern on cardiac magnetic resonance and light chains
    (2023) SLIVNICK, Jeremy A.; ALVI, Nazia; SINGULANE, Cristiane C.; SCHEETZ, Seth; GOYAL, Akash; PATEL, Hena; SARSWAT, Nitasha; ADDETIA, Karima; FERNANDES, Fabio; VIEIRA, Marcelo Luiz Campos; CAFEZEIRO, Caio Reboucas Fonseca; CARVALHAL, Suenia Freitas; SIMONETTI, Orlando P.; SINGH, Jai; LANG, Roberto M.; ZAREBA, Karolina M.; PATEL, Amit R.
    Aims While cardiac magnetic resonance (CMR) is often obtained early in the evaluation of suspected cardiac amyloidosis (CA), it currently cannot be utilized to differentiate immunoglobulin (AL) and transthyretin (ATTR) CA. We aimed to determine whether a novel CMR and light-chain biomarker-based algorithm could accurately diagnose ATTR-CA. Methods and results Patients with confirmed AL or ATTR-CA with typical late gadolinium enhancement (LGE) and Look-Locker pattern for CA on CMR were retrospectively identified at three academic medical centres. Comprehensive light-chain analysis including free light chains, serum, and urine electrophoresis/immunofixation was performed. The diagnostic accuracy of the typical CMR pattern for CA in combination with negative light chains for the diagnosis of ATTR-CA was determined both in the entire cohort and in the subset of patients with invasive tissue biopsy as the gold standard. A total of 147 patients (age 70 +/- 11, 76% male, 51% black) were identified: 89 ATTR-CA and 58 AL-CA. Light-chain biomarkers were abnormal in 81 (55%) patients. Within the entire cohort, the sensitivity and specificity of a typical LGE and Look-Locker CMR pattern and negative light chains for ATTR-CA was 73 and 98%, respectively. Within the subset with biopsy-confirmed subtype, the CMR and light-chain algorithm were 69% sensitive and 98% specific. Conclusion The combination of a typical LGE and Look-Locker pattern on CMR with negative light chains is highly specific for ATTR-CA. The successful non-invasive diagnosis of ATTR-CA using CMR has the potential to reduce diagnostic and therapeutic delays and healthcare costs for many patients.