BRUNO VAZ KERGES BUENO

(Fonte: Lattes)
Índice h a partir de 2011
2
Lattes
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Instituto do Coração, Hospital das Clínicas, Faculdade de Medicina - Médico
LIM/11 - Laboratório de Cirurgia Cardiovascular e Fisiopatologia da Circulação, Hospital das Clínicas, Faculdade de Medicina

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  • conferenceObject
    Myocardial Stiffness evaluation by shear wave elastography in transthyretin amyloidosis with and without cardiac involvement
    (2023) ALENCAR NETO, A. C. Comte de; CAFEZEIRO, C. R. F. C. Caio Reboucas Fonseca; BUENO, B. V. K. B. Bruno Vaz Kerges; RISSATO, J. H. R. Joao Henrique; HOTTA, V. T. H. Viviane Tiemi; ALENCAR FILHO, A. C. A. F. Aristoteles Comte De; PEREIRA, F. L. P. Fernando Linhares; COUCEIRO, K. C. Katia; ROCHITTE, C. E. R. Carlos Eduardo; BARBOSA, J. M. B. B. Joao Marcos Bemfica; SOARES JUNIOR, J. S. J. Jose; RAMIRES, F. J. A. R. Felix Jose Alvarez; KALIL FILHO, R. K. F. Roberto; MATHIAS JUNIOR, W. M. J. Wilson; FERNANDES, F. F. Fabio
  • conferenceObject
    Coronary flow reserve by PET 13N-ammonia in patients with hereditary transthyretin amyloidosis with and without cardiac involvement
    (2022) ALENCAR NETO, A. C.; CAFEZEIRO, C. R. F.; BUENO, B. V. K.; SOUZA, F. Ribeiro De; RISSATO, J. H. S. Henrique; BORGES, T. Souza; CARVALHAL, S. Freitas; LIMA, M. Santos; BUCHPIGUEL, C. Alberto; CHALELA, W. Azem; RAMIRES, F. J. Alvarez; SZOR, R. Shcolnik; KALIL FILHO, R.; ROCHITTE, C. E.; FERNANDES, F.
  • article 3 Citação(ões) na Scopus
    Amyloid Cardiomyopathy Establishing a Patient Referral Center in Brazil
    (2021) SZOR, Roberta Shcolnik; ALENCAR, Aristoteles; BUENO, Bruno Vaz Kerges; CAFEZEIRO, Caio Reboucas Fonseca; ALVES, Lucas Bassolli de Oliveira; HAJJAR, Ludhmila Abrahao; MARTINEZ, Gracia Aparecida; ROCHA, Vanderson; FERNANDES, Fabio
  • conferenceObject
    Non-invasive assessment of myocardial stiffness by the two-dimensional shear wave elastography ultrasound technique in patients with amyloidosis and Fabry disease
    (2023) CAFEZEIRO, C.; NETO, A. C. Alencar; BUENO, B. V. K.; RISSATO, J. H.; PEREIRA, N. M.; PEREIRA, F. L.; RAMIRES, F. J. A.; MATHIAS JR., W.; ROCHITTE, C. E.; HOTTA, V. T.; DABARIAN, A. L.; FERNANDES, F.
  • bookPart 0 Citação(ões) na Scopus
    Cardiologic Manifestation in Amyloidosis
    (2023) ALENCAR NETO, A. C. de; CAFEZEIRO, C. R. F.; BUENO, B. V. K.; SINGULANE, C. de Carvalho; FERNANDES, F.
    ATTR and AL amyloidosis are responsible for approximately 95% of all cases of CA. Cardiac amyloidosis (CA) is a relatively common cause of heart failure with preserved ejection fraction (HFpEF). It is important to know that extracardiac changes may precede cardiac involvement by a few years; thus, it is essential to recognize these findings as part of the clinical feature in a patient with amyloidosis. CA results from the progressive deposition of amyloid fibrils in the extracellular space. The involvement of amyloidosis includes mechanical and electrical manifestations. The most frequent CA clinical presentation is HFpEF with a restrictive pattern leading to diastolic dysfunction, followed by systolic dysfunction. Conduction disorders and arrhythmias are commonly associated. © The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerland AG 2023.
  • article 4 Citação(ões) na Scopus
    Clinical, Laboratory, and Imaging Profile in Patients with Systemic Amyloidosis in a Brazilian Cardiology Referral Center
    (2022) FERNANDES, Fabio; ALENCAR NETO, Aristoteles Comte de; BUENO, Bruno Vaz Kerges; CAFEZEIRO, Caio Reboucas Fonseca; RISSATO, Joao Henrique; SZOR, Roberta Shcolnik; CARVALHO, Mariana Lombardi Peres de; MATHIAS JUNIOR, Wilson; LINO, Angelina Maria Martins; CASTELLI, Jussara Bianchi; SOUZA, Evandro de Oliveira; RAMIRES, Felix Jose Alvarez; HOTTA, Viviane Tiemi; SOARES JUNIOR, Jose; TAVARES, Caio de Assis Moura; KRIEGER, Jose Eduardo; ROCHITTE, Carlos Eduardo; DABARIAN, Andre; HAJJAR, Ludhmila Abrahao; KALIL FILHO, Roberto; MADY, Charles
    Background: Systemic amyloidosis is a disease with heterogeneous clinical manifestations. Diagnosis depends on clinical suspicion combined with specific complementary methods. Objective: To describe the clinical, laboratory, electrocardiographic, and imaging profile in patients with systemic amyloidosis with cardiac involvement. Methods: This study was conducted with a convenience sample, analyzing clinical, laboratory, electrocardiographic, echocardiographic, nuclear medicine, and magnetic resonance data. Statistical significance was set at p < 0.05. Results: A total of 105 patients were evaluated (median age of 66 years), 62 of whom were male. Of all patients, 83 had transthyretin (ATTR) amyloidosis, and 22 had light chain (AL) amyloidosis. With respect to ATTR cases, 68.7% were the hereditary form (ATTRh), and 31.3% were wild type (ATTRw). The most prevalent mutations were Val142Ile (45.6%) and Val50Met (40.3%). Time from onset of symptoms to diagnosis was 0.54 and 2.15 years, in the AL and ATTR forms, respectively (p < 0.001). Cardiac involvement was observed in 77.9% of patients with ATTR and in 90.9% of those with AL. Alterations were observed in atrioventricular and intraventricular conduction in 20% and 27.6% of patients, respectively, with 33.7% in ATTR and 4.5% in AL (p = 0.006). In the ATTRw form, there were more atrial arrhythmias than in ATTRh (61.5% versus 22.8%; p = 0.001). On echocardiogram, median septum thickness in ATTRw, ATTRh, and AL was 15 mm, 12 mm, and 11 mm, respectively (p = 0.193). Elevated BNP was observed in 89.5% of patients (median 249, ICR 597.7), and elevated troponin was observed in 43.2%. Conclusion: In this setting, it was possible to characterize cardiac involvement in systemic amyloidosis in its different subtypes by means of clinical history and the diagnostic methods described.